Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
Results 1-3 of 88
Page 36
... specific lectins of Ulex euro- paeus ( UEA - 1 ) bind selectively to M cells of Peyer's patches , 64 , 65 and galactose lectins bind to colonic and rectal M cells.64 In rabbit cecal patches , lectins specific for fucose and N ...
... specific lectins of Ulex euro- paeus ( UEA - 1 ) bind selectively to M cells of Peyer's patches , 64 , 65 and galactose lectins bind to colonic and rectal M cells.64 In rabbit cecal patches , lectins specific for fucose and N ...
Page 250
... specific genetic markers , and most recent linkages between specific chromosomal regions and IBD in families . It is the principal thesis of this chapter that the inflammatory bowel diseases fundamentally are genetic diseases with ...
... specific genetic markers , and most recent linkages between specific chromosomal regions and IBD in families . It is the principal thesis of this chapter that the inflammatory bowel diseases fundamentally are genetic diseases with ...
Page 294
... specific immune re- sponses by feeding protein antigens . IV . Evidence for tolerance and specific active suppression of cell - mediated immune responses to ovalbumin . J Immunol 1979 ; 123 : 2344 . 76. Mattingly JA , Waksman BH ...
... specific immune re- sponses by feeding protein antigens . IV . Evidence for tolerance and specific active suppression of cell - mediated immune responses to ovalbumin . J Immunol 1979 ; 123 : 2344 . 76. Mattingly JA , Waksman BH ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis