The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 14
... CORTISOL BIOSYNTHESIS WITHOUT METOPIRONE ( METYRAPONE ) ADMINISTRATION PITUITARY NORMAL ACTH NORMAL BLOCK CORTISOL CORTISOL NORMAL COMP . S tter M.D. OCIBA 17 - HYDROXY- CORTICOIDS NORMAL IN PLASMA AND TEST FOR ADRENAL CORTICAL HORMONE ...
... CORTISOL BIOSYNTHESIS WITHOUT METOPIRONE ( METYRAPONE ) ADMINISTRATION PITUITARY NORMAL ACTH NORMAL BLOCK CORTISOL CORTISOL NORMAL COMP . S tter M.D. OCIBA 17 - HYDROXY- CORTICOIDS NORMAL IN PLASMA AND TEST FOR ADRENAL CORTICAL HORMONE ...
Page 83
... CORTISOL CORTISOL 11 CORTISOL ALBUMIN ANGIOTENSIN II LOW BLOOD VOLUME HIGH POTASSIUM LOW SODIUM ALDOSTERONE 17 - KETOSTEROIDS L LIVER KIDNEY RENIN behaves more like cortisol and is a less potent sodium container than aldosterone . The ...
... CORTISOL CORTISOL 11 CORTISOL ALBUMIN ANGIOTENSIN II LOW BLOOD VOLUME HIGH POTASSIUM LOW SODIUM ALDOSTERONE 17 - KETOSTEROIDS L LIVER KIDNEY RENIN behaves more like cortisol and is a less potent sodium container than aldosterone . The ...
Page 84
... CORTISOL In man , the most abundant glucocorti- coids are cortisol and cortisone . Their various physiological actions , when exag- gerated by excessively high levels , will lead to Cushing's disease ( see page 85 ) . Glucocorticoids ...
... CORTISOL In man , the most abundant glucocorti- coids are cortisol and cortisone . Their various physiological actions , when exag- gerated by excessively high levels , will lead to Cushing's disease ( see page 85 ) . Glucocorticoids ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin