The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 31
... Cushing's syn- drome , although it is now agreed that the clinical features of the syndrome result from hyperfunction of the adrenal cortex . Cushing considered that the dis- order was due primarily to a basophil adenoma of the anterior ...
... Cushing's syn- drome , although it is now agreed that the clinical features of the syndrome result from hyperfunction of the adrenal cortex . Cushing considered that the dis- order was due primarily to a basophil adenoma of the anterior ...
Page 85
... CUSHING'S SYNDROME Clinical Findings This syndrome ( or disease ) carries the name of the Boston neurosurgeon , Harvey Cushing , who first described its signs and symptoms and linked it to a basophilic adenoma of the anterior pituitary ...
... CUSHING'S SYNDROME Clinical Findings This syndrome ( or disease ) carries the name of the Boston neurosurgeon , Harvey Cushing , who first described its signs and symptoms and linked it to a basophilic adenoma of the anterior pituitary ...
Page 89
... syndrome is induced by increased amounts of some type of corticotropin of either pituitary or other origin ( see page 231 ) . Since Cushing's original descrip- tion , the rĂ´le of the pituitary in the path- ogenesis of adrenal ...
... syndrome is induced by increased amounts of some type of corticotropin of either pituitary or other origin ( see page 231 ) . Since Cushing's original descrip- tion , the rĂ´le of the pituitary in the path- ogenesis of adrenal ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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Common terms and phrases
abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin