The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 37
... activity ; ( 2 ) no change in ATP , P32 uptake , and mon- amine oxidase activity ; and ( 3 ) increase in 5 - hydroxytryptophan decarboxylase activity . Light also produces other changes in the female rat : ( 1 ) decrease in adrenal size ...
... activity ; ( 2 ) no change in ATP , P32 uptake , and mon- amine oxidase activity ; and ( 3 ) increase in 5 - hydroxytryptophan decarboxylase activity . Light also produces other changes in the female rat : ( 1 ) decrease in adrenal size ...
Page 108
... activity begins to lag behind . In attempts to eliminate or reduce the catabolic effect as well as the sodium - retaining activity of cortisol , numerous derivatives have been synthe- sized , some of which are shown below . Relative ...
... activity begins to lag behind . In attempts to eliminate or reduce the catabolic effect as well as the sodium - retaining activity of cortisol , numerous derivatives have been synthe- sized , some of which are shown below . Relative ...
Page 182
... activity and excessive resorption of cal- cium and phosphate from the bone minerals ( dahllite or hydroxyapatite ) . The presence of chronic acidosis in renal failure enhances this bone resorptive activity . The resulting subperiosteal ...
... activity and excessive resorption of cal- cium and phosphate from the bone minerals ( dahllite or hydroxyapatite ) . The presence of chronic acidosis in renal failure enhances this bone resorptive activity . The resulting subperiosteal ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin