The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 152
... ACID FAT CELL GLYCOGEN FORMATION AND BREAKDOWN GLUCOSE- 6 - PHOSPHATE. PROTEIN - CARBOHYDRATE INTERRELATIONSHIPS Free amino acids are present in the circulation , in the extracellular fluids , and in the cell interior . They are derived ...
... ACID FAT CELL GLYCOGEN FORMATION AND BREAKDOWN GLUCOSE- 6 - PHOSPHATE. PROTEIN - CARBOHYDRATE INTERRELATIONSHIPS Free amino acids are present in the circulation , in the extracellular fluids , and in the cell interior . They are derived ...
Page 218
A Compilation of Pathological and Anatomical Paintings Prepared Frank Henry Netter. EACH ACTIVATED AMINO ACID IS ... AMINO ACIDS . EACH TRANSPORT RNA IS " CODED " BY ITS BASES τα DNA ( MILLIONS OF BASES LONG ) ADDI MESSENGER. MITOCHONDRION ...
A Compilation of Pathological and Anatomical Paintings Prepared Frank Henry Netter. EACH ACTIVATED AMINO ACID IS ... AMINO ACIDS . EACH TRANSPORT RNA IS " CODED " BY ITS BASES τα DNA ( MILLIONS OF BASES LONG ) ADDI MESSENGER. MITOCHONDRION ...
Page 219
... AMINO ACID COMBINATION TO THE MESSENGER . THE AMINO ACIDS FORM PEPTIDE BONDS . THE FIRST RNA IS RELEASED THE COMPLETED PROTEIN IS RELEASED . THE RIBOSOME IS FREED FOR REUSE region of the t - RNA chain to the codon on the messenger . The ...
... AMINO ACID COMBINATION TO THE MESSENGER . THE AMINO ACIDS FORM PEPTIDE BONDS . THE FIRST RNA IS RELEASED THE COMPLETED PROTEIN IS RELEASED . THE RIBOSOME IS FREED FOR REUSE region of the t - RNA chain to the codon on the messenger . The ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin