The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 177
... CHIEF CELLS ; B = OXYPHIL CELLS PAS STAIN , X 675 GLYCOGEN IN CHIEF CELLS ULTRASTRUCTURE OF PARATHYROID GLAND ... cell ( inactive cell ) measures approximately 8 microns in diameter . It has a well - defined cell mem- brane and a 4- to 5 ...
... CHIEF CELLS ; B = OXYPHIL CELLS PAS STAIN , X 675 GLYCOGEN IN CHIEF CELLS ULTRASTRUCTURE OF PARATHYROID GLAND ... cell ( inactive cell ) measures approximately 8 microns in diameter . It has a well - defined cell mem- brane and a 4- to 5 ...
Page 179
... CELL HYPERPLASIA ABOUT 10 % OF CASES PRIMARY CHIEF CELL HYPERPLASIA ABOUT 8 % OF CASES CARCINOMA ABOUT 2 % OF CASES PATHOLOGIC PHYSIOLOGY OF PRIMARY HYPERPARATHYROIDISM Primary hyperparathyroidism is most commonly due to a single ...
... CELL HYPERPLASIA ABOUT 10 % OF CASES PRIMARY CHIEF CELL HYPERPLASIA ABOUT 8 % OF CASES CARCINOMA ABOUT 2 % OF CASES PATHOLOGIC PHYSIOLOGY OF PRIMARY HYPERPARATHYROIDISM Primary hyperparathyroidism is most commonly due to a single ...
Page 183
... CELLS AND CHIEF CELLS IN ADENOMA ; H. AND E. STAIN , X 35 ( involving 2 glands ) 13 ( 4 % ) Carcinoma . II ( 4 % ) MONONUCLEAR GIANT CELLS Hyperplasia 36 ( 12 % ) Clear Cell 15 ( 5 % ) PRIMARY HYPERPLASIA : CHIEF CELL 300 ( 100 % ) Chief ...
... CELLS AND CHIEF CELLS IN ADENOMA ; H. AND E. STAIN , X 35 ( involving 2 glands ) 13 ( 4 % ) Carcinoma . II ( 4 % ) MONONUCLEAR GIANT CELLS Hyperplasia 36 ( 12 % ) Clear Cell 15 ( 5 % ) PRIMARY HYPERPLASIA : CHIEF CELL 300 ( 100 % ) Chief ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin