The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 54
... excessive secretion of LATS , with , possibly , an increased sensitivity of the thyroid gland to TSH , accounts for the excessive thyroid hormone secretion . The possibility that , in certain cases , a spontaneous , diffuse overactivity ...
... excessive secretion of LATS , with , possibly , an increased sensitivity of the thyroid gland to TSH , accounts for the excessive thyroid hormone secretion . The possibility that , in certain cases , a spontaneous , diffuse overactivity ...
Page 131
... EXCESSIVE SECRETING TUMOR CHROMOSOMAL ABNORMALITY 44 44 ORAL OR PARENTERAL + + Xo XY ADMINISTRATION PLACENTA MOSAIC OR MALE CHROMOSOMAL PATTERN HIRSUTISM AND VIRILIZATION The causes of hirsutism are many and diverse , from a racial or ...
... EXCESSIVE SECRETING TUMOR CHROMOSOMAL ABNORMALITY 44 44 ORAL OR PARENTERAL + + Xo XY ADMINISTRATION PLACENTA MOSAIC OR MALE CHROMOSOMAL PATTERN HIRSUTISM AND VIRILIZATION The causes of hirsutism are many and diverse , from a racial or ...
Page 242
... excessive deposition of oxalates in the system . The common condition of calcium oxalate stones is usually associated with normal amounts of oxalic acid in urine and tis- sues . The much rarer hereditary , recessive disease , primary ...
... excessive deposition of oxalates in the system . The common condition of calcium oxalate stones is usually associated with normal amounts of oxalic acid in urine and tis- sues . The much rarer hereditary , recessive disease , primary ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin