The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
From inside the book
Results 1-3 of 30
Page 86
... PIGMENTATION BOTH ADRENAL CORTICES OVERACTIVE- AND / OR HYPERPLASTIC- EXCESS HYPOTHALAMIC STIMULATION- INCREASED BASOPHIL ACTIVITY SELLA TURCICA NORMAL CHROMOPHOBE ADENOMA OF PITUITARY SELLA TURCICA- ENLARGED CORTISOL 17 - KETOSTEROIDS ...
... PIGMENTATION BOTH ADRENAL CORTICES OVERACTIVE- AND / OR HYPERPLASTIC- EXCESS HYPOTHALAMIC STIMULATION- INCREASED BASOPHIL ACTIVITY SELLA TURCICA NORMAL CHROMOPHOBE ADENOMA OF PITUITARY SELLA TURCICA- ENLARGED CORTISOL 17 - KETOSTEROIDS ...
Page 89
... PIGMENTATION Most of the clinical features of Cush- ing's syndrome are direct consequences of adrenal cortical hyperfunction , which produces a sustained excess of glucocor- ticoid hormones , chiefly cortisol . In the absence of an ...
... PIGMENTATION Most of the clinical features of Cush- ing's syndrome are direct consequences of adrenal cortical hyperfunction , which produces a sustained excess of glucocor- ticoid hormones , chiefly cortisol . In the absence of an ...
Page 101
... PIGMENTATION- SKIN PIGMENTATION DARKENING OF HAIR FRECKLING- VITILIGO PIGMENT ACCENTUATION AT NIPPLES , AT FRICTION AREAS PIGMENT CONCENTRATION IN SKIN CREASES AND IN SCARS- ₤ Netter M.D. CIBA tion . Assessment of pigmentation requires ...
... PIGMENTATION- SKIN PIGMENTATION DARKENING OF HAIR FRECKLING- VITILIGO PIGMENT ACCENTUATION AT NIPPLES , AT FRICTION AREAS PIGMENT CONCENTRATION IN SKIN CREASES AND IN SCARS- ₤ Netter M.D. CIBA tion . Assessment of pigmentation requires ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
20 other sections not shown
Other editions - View all
Common terms and phrases
abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin