The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page xiv
... ( Rickets ) of Alimentary Origin 190 189 15. Osteomalacia ( Rickets ) of Renal Origin 16. Clinical Manifestations of Osteomalacia 191 14. Hormonal Treatment of Cancer of the Breast 232 15. Hormonal Treatment of Various Cancers SECTION IX ...
... ( Rickets ) of Alimentary Origin 190 189 15. Osteomalacia ( Rickets ) of Renal Origin 16. Clinical Manifestations of Osteomalacia 191 14. Hormonal Treatment of Cancer of the Breast 232 15. Hormonal Treatment of Various Cancers SECTION IX ...
Page 182
... rickets ) is the usual event in any patient showing chronic renal insufficiency of any kind , with diminished glomerular filtration . Aside from retaining nonprotein nitrogen ( NPN ) , including waste products such as creatinine , which ...
... rickets ) is the usual event in any patient showing chronic renal insufficiency of any kind , with diminished glomerular filtration . Aside from retaining nonprotein nitrogen ( NPN ) , including waste products such as creatinine , which ...
Page 191
... ( RICKETS ) OF RENAL ORIGIN The second large category in osteo- malacia or rickets ( referred to on page 190 ) appears to be associated primarily with excessive renal losses of calcium and / or phosphate ( renal rickets , vitamin- D ...
... ( RICKETS ) OF RENAL ORIGIN The second large category in osteo- malacia or rickets ( referred to on page 190 ) appears to be associated primarily with excessive renal losses of calcium and / or phosphate ( renal rickets , vitamin- D ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin