The Ciba Collection of Medical Illustrations;: A Compilation of Pathological and Anatomical Paintings Prepared, Volume 4 |
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Page 31
... SYNDROME ( MAY BE SYMPTOM - FREE ) OXYTOCIN MIGRATES ALONG NERVE FIBERS. SECTION I - PLATE 29 BASOPHIL ADENOMA MINUTE ADENOMA BASOPHIL ADENOMA OF MODERATE SIZE ; CHARACTERISTICALLY NO ENLARGEMENT OF SELLA TURCICA #Nitter OCIBA Basophil ...
... SYNDROME ( MAY BE SYMPTOM - FREE ) OXYTOCIN MIGRATES ALONG NERVE FIBERS. SECTION I - PLATE 29 BASOPHIL ADENOMA MINUTE ADENOMA BASOPHIL ADENOMA OF MODERATE SIZE ; CHARACTERISTICALLY NO ENLARGEMENT OF SELLA TURCICA #Nitter OCIBA Basophil ...
Page 89
... syndrome are direct consequences of adrenal cortical hyperfunction , which produces a sustained excess of glucocor- ticoid hormones , chiefly cortisol . In the absence of an adrenal tumor and in the presence of hyperplastic adrenal ...
... syndrome are direct consequences of adrenal cortical hyperfunction , which produces a sustained excess of glucocor- ticoid hormones , chiefly cortisol . In the absence of an adrenal tumor and in the presence of hyperplastic adrenal ...
Page 187
... syndrome resembling idiopathic hypoparathyroidism , but having asso- ciated genetic defects such as short stat- ure , round face , short metacarpals , and ectopic bone formation , is called pseudo- hypoparathyroidism . In this syndrome ...
... syndrome resembling idiopathic hypoparathyroidism , but having asso- ciated genetic defects such as short stat- ure , round face , short metacarpals , and ectopic bone formation , is called pseudo- hypoparathyroidism . In this syndrome ...
Contents
SECTION | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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abnormal absence acromegaly ACTH action activity adenoma administration adrenal adrenal cortical adult aldosterone amino acids amounts androgens appear artery associated become blood body bone calcium cancer cause cells cent changes characteristic cholesterol chromosome CIBA clinical common Continued cortex cortisol decrease deficiency diabetes disease early effect elevated enlargement enzyme estrogen excessive excretion female formation frequently function gland glucose gonadal growth homogentisic acid hormone human hyperplasia HYPOTHYROIDISM increased individuals inhibition insufficiency insulin involved iodine kidney lateral lead lesions less liver loss lower male marked metabolism muscle nerve Netter normal obesity occur OCIBA parathyroid patients phosphate pituitary plasma PLATE present primary produce protein rare reduced renal response result rickets rise secretion seen serum severe specific STAIN stimulation structures studies sugar suprarenal syndrome thyroid tion tissue tumor urinary urine usually vitamin