CNS Injuries: Cellular Responses and Pharmacological StrategiesThe basic science of the cellular and molecular responses of the brain to injury is a rapidly expanding area of research that provides evidence of growing opportunities for pharmacological intervention in the clinic. CNS Injuries: Cellular Responses and Pharmacological Strategies is an up-to-date examination of new developments in our understanding of the cellular and trophic responses to CNS injuries and the potential treatment. This text collates reviews of the most important areas of study regarding injury response including Providing a record of recent advances that will help point the way to future developments, this enlightening reference is sure to benefit researchers and practitioners in a broad range of disciplines, including: neurology, pharmacology, pathology, toxicology, immunology, and many others. |
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CNS Injuries: Cellular Responses and Pharmacological Strategies Martin Berry,Ann Logan No preview available - 1998 |
Common terms and phrases
Acad activated microglia adhesion molecules adult rat antibodies antigen astrocytes autoimmune axons axotomy BDNF binding Biol blood blood-brain barrier brain macrophages Brain Res cell death cellular central nervous system cerebral cholinergic cholinergic neurons CNS injury CNTF cultured cytokines damage demyelination disease dopaminergic effects endothelial cells endothelium enhanced expression fibroblasts function GDNF glia glial cells glutamate hippocampal human immune response Immunol increase induced inflammation inflammatory infusion inhibition inhibitors injection integrins interaction ischaemia ischaemic isoforms Kreutzberg L-selectin lesion leucocyte ligand lymphocytes macrophages mediated membrane microglia microglial activation microglial cells migration monocytes morphology mRNA multiple sclerosis myelin neonatal nerve growth factor neurite Neuroimmunol neurons Neuropathol Neurosci neurotrophic factors neutrophils normal oligodendrocytes optic nerve perivascular phagocytic proliferation promote proteases protein rat brain receptor release retinal role scar specific spinal cord studies survival TGF-B TGF-ẞ tissue transection transforming growth upregulation vascular vitro vivo wound