The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 47
... bilirubin in the blood results in its appearance in the urine . Most of the hemoglobin molecule ( 96 per cent ) for each species is globin , a specific protein to which the pigment radicle , heme , is attached . Heme con- sists of four ...
... bilirubin in the blood results in its appearance in the urine . Most of the hemoglobin molecule ( 96 per cent ) for each species is globin , a specific protein to which the pigment radicle , heme , is attached . Heme con- sists of four ...
Page 48
... BILIRUBIN O UROBILINOGEN ++++ -JAUNDICE LIGHT REDDISH YELLOW ( PLUS ANEMIC PALLOR ) BILIVERDINGLOBIN INDIRECT BILIRUBIN ( UNCONJUGATED ) PROMPT - REACTING BILIRUBIN ( CONJUGATED ) UROBILINOGEN BOWEL STOOL DARK ALL THESE OCCUR IN LIVER ...
... BILIRUBIN O UROBILINOGEN ++++ -JAUNDICE LIGHT REDDISH YELLOW ( PLUS ANEMIC PALLOR ) BILIVERDINGLOBIN INDIRECT BILIRUBIN ( UNCONJUGATED ) PROMPT - REACTING BILIRUBIN ( CONJUGATED ) UROBILINOGEN BOWEL STOOL DARK ALL THESE OCCUR IN LIVER ...
Page 180
... BILIRUBIN UNCONJUGATED BILIRUBIN CONGENITAL AND FAMILIAL HYPERBILIRUBINEMIAS - Most benign hyperbilirubinemias are congenital and frequently familial . For- merly , they were designated as retention jaundice ( see page 48 ) , but they ...
... BILIRUBIN UNCONJUGATED BILIRUBIN CONGENITAL AND FAMILIAL HYPERBILIRUBINEMIAS - Most benign hyperbilirubinemias are congenital and frequently familial . For- merly , they were designated as retention jaundice ( see page 48 ) , but they ...
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The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis