The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 40
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
Page 77
Frank Henry Netter. LIPOGENIC FACTORS 1 IMPACTED STONE WITH EDEMA OBSTRUCTIVE JAUNDICE DEVELOPS ONLY IF. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT METHIONINE DECREASED METABOLIC NEEDS PROTEIN LIPOTROPIC FACTORS ...
Frank Henry Netter. LIPOGENIC FACTORS 1 IMPACTED STONE WITH EDEMA OBSTRUCTIVE JAUNDICE DEVELOPS ONLY IF. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT METHIONINE DECREASED METABOLIC NEEDS PROTEIN LIPOTROPIC FACTORS ...
Page 79
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
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The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis