The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 7
... glycogen pro- duces a fine , granulated and vacuolated appearance of the cytoplasm . In severe disease of any kind , particularly in the agonal period , the glycogen content becomes markedly reduced , so that , as a rule , in autopsy ...
... glycogen pro- duces a fine , granulated and vacuolated appearance of the cytoplasm . In severe disease of any kind , particularly in the agonal period , the glycogen content becomes markedly reduced , so that , as a rule , in autopsy ...
Page 36
... glycogen . Hepatic glycogen is the main form of carbohydrate storage . The process of glycogen synthesis is called glycogenesis ; the breakdown of glycogen to liberate glucose , thereby maintaining the blood sugar level , is ...
... glycogen . Hepatic glycogen is the main form of carbohydrate storage . The process of glycogen synthesis is called glycogenesis ; the breakdown of glycogen to liberate glucose , thereby maintaining the blood sugar level , is ...
Page 86
... glycogen storage or Gierke's disease , an inborn error of the metabolism , exces- sive quantities of glycogen accumulate in the liver cells , which become enlarged and exhibit a finely vacuolized or granu- lated cytoplasm with a central ...
... glycogen storage or Gierke's disease , an inborn error of the metabolism , exces- sive quantities of glycogen accumulate in the liver cells , which become enlarged and exhibit a finely vacuolized or granu- lated cytoplasm with a central ...
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The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis