The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 48
... Increased production or decreased excretion of biliary pigments leads to jaundice without bile flow impairment . Hemolytic jaun- dice is due to overproduction of bile pigment . It occurs in septicemia , pernicious anemia , acquired or ...
... Increased production or decreased excretion of biliary pigments leads to jaundice without bile flow impairment . Hemolytic jaun- dice is due to overproduction of bile pigment . It occurs in septicemia , pernicious anemia , acquired or ...
Page 74
... increased and reabsorption of tissue fluid is im- paired . If hypoproteinemia complicates portal hypertension , a rapid development . of ascites may be expected and occurs promptly , e.g. , after hemorrhage from ruptured esophageal ...
... increased and reabsorption of tissue fluid is im- paired . If hypoproteinemia complicates portal hypertension , a rapid development . of ascites may be expected and occurs promptly , e.g. , after hemorrhage from ruptured esophageal ...
Page 171
... INCREASED IN KUPFFER CELLS MORE MOBILIZED KUPFFER CELLS- TISSUE SPACE WIDENED ; CONTAINS MANY COLLAGEN FIBRILS AND DEBRIS MICROVILLI IRREGULAR- PINOCYTOTIC DROPLETS INCREASED- FAT DROPLETS INCREASED- MITOCHONDRIA SWOLLEN , IRREGULAR ...
... INCREASED IN KUPFFER CELLS MORE MOBILIZED KUPFFER CELLS- TISSUE SPACE WIDENED ; CONTAINS MANY COLLAGEN FIBRILS AND DEBRIS MICROVILLI IRREGULAR- PINOCYTOTIC DROPLETS INCREASED- FAT DROPLETS INCREASED- MITOCHONDRIA SWOLLEN , IRREGULAR ...
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The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis