The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 41
... INTESTINE 3. TAKEN UP BY LIVER A , B , C POSSIBLE POINTS OF BLOCKAGE INHIBITS CONVERSION OF HEXOSE TO GLYCOGEN FACILITATES INTESTINAL ABSORPTION OF HEXOSE ORAL 4 . EXCESS EXCRETED BY KIDNEYS facilitates intestinal glucose absorption ...
... INTESTINE 3. TAKEN UP BY LIVER A , B , C POSSIBLE POINTS OF BLOCKAGE INHIBITS CONVERSION OF HEXOSE TO GLYCOGEN FACILITATES INTESTINAL ABSORPTION OF HEXOSE ORAL 4 . EXCESS EXCRETED BY KIDNEYS facilitates intestinal glucose absorption ...
Page 88
... intestinal mucosa . There it is reoxidized , and the ferric iron is bound to a protein , apoferritin . The iron - protein complex , ferritin , is the main organic storage form of iron but before iron is released to the blood , ferritin ...
... intestinal mucosa . There it is reoxidized , and the ferric iron is bound to a protein , apoferritin . The iron - protein complex , ferritin , is the main organic storage form of iron but before iron is released to the blood , ferritin ...
Page 102
... intestinal disease , but it involves the liver in one tenth to one third of the cases . The infec- tive , encysted form of the parasite , com- municated from man to man , without an intermediate host , by water , food , etc. , which has ...
... intestinal disease , but it involves the liver in one tenth to one third of the cases . The infec- tive , encysted form of the parasite , com- municated from man to man , without an intermediate host , by water , food , etc. , which has ...
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The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis