The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
From inside the book
Results 1-3 of 36
Page 68
... lobular periph- ery , and , eventually , to regenerative nodules . The septa fuse eventually to connect portal tracts and central fields . These connections contain vessels which become portal vein - hepatic vein anasto- moses . Liver ...
... lobular periph- ery , and , eventually , to regenerative nodules . The septa fuse eventually to connect portal tracts and central fields . These connections contain vessels which become portal vein - hepatic vein anasto- moses . Liver ...
Page 94
... lobular architecture may be exaggerated , with dark - red depressed CUT SURFACE : ACUTELY CONGESTED , " SPLEENLIKE " LOW MAGNIFICATION : MASSIVE NECROSIS , INFILTRATION OF LOBULES AND PORTAL AREAS , BILE DUCT PROLIFERATION and enlarged ...
... lobular architecture may be exaggerated , with dark - red depressed CUT SURFACE : ACUTELY CONGESTED , " SPLEENLIKE " LOW MAGNIFICATION : MASSIVE NECROSIS , INFILTRATION OF LOBULES AND PORTAL AREAS , BILE DUCT PROLIFERATION and enlarged ...
Page 110
... lobular fat accumulation . Hemolysis may cause increased iron as well as bile pig- mentation of the Kupffer cells ... lobular parenchyma and of mesenchymal cells in the portal triad , granulomas develop which coalesce and become necrotic ...
... lobular fat accumulation . Hemolysis may cause increased iron as well as bile pig- mentation of the Kupffer cells ... lobular parenchyma and of mesenchymal cells in the portal triad , granulomas develop which coalesce and become necrotic ...
Other editions - View all
The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis