The Ciba Collection of Medical Illustrations, Volume 3, Part 3Ciba [Pharmaceutical Products, 1972 - Anatomy, Pathological |
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Page 64
... NECROSIS Necrosis implies not only death of cells but also the phenomena following cell death , namely , the disappearance of cells and frequently also the ... NECROSIS MASSIVE NECROSIS MASSIVE COLLAPSE CENTRAL NECROSIS B SUBMASSIVE 64.
... NECROSIS Necrosis implies not only death of cells but also the phenomena following cell death , namely , the disappearance of cells and frequently also the ... NECROSIS MASSIVE NECROSIS MASSIVE COLLAPSE CENTRAL NECROSIS B SUBMASSIVE 64.
Page 95
... necrosis , instead of seizing more or less simultaneously all lobules , as in acute fulminant hepatitis ( see page 94 ) , is restricted to only some lobules at one time but proceeds at intervals from one part of the liver to another ...
... necrosis , instead of seizing more or less simultaneously all lobules , as in acute fulminant hepatitis ( see page 94 ) , is restricted to only some lobules at one time but proceeds at intervals from one part of the liver to another ...
Page 143
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
Other editions - View all
The Ciba Collection of Medical Illustrations: A Compilation of Paintings on ... Frank Henry Netter No preview available - 1959 |
Common terms and phrases
abdominal abnormal abscesses acid acute albumin Amer amino amylase anastomoses Anatomy appear ascites biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholelithiasis cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts damage degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal excretion extrahepatic fibrosis fistula formation frequently function gallbladder gland glycogen hemochromatosis hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein hepatocellular infection inferior injury intestinal intrahepatic jaundice Kupffer cells left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism metastases necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct parenchyma patients peritoneal phosphatase pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules result right hepatic septa serum sinusoids sphincter spleen splenic stones SUPERIOR MESENTERIC surface surgical tion tumor urobilinogen usually vascular vena cava vessels viral hepatitis