The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 47
SECTION XVI — PLATE I 2 BILE PIGMENT METABoLISM Normal and Abnormal
From the quantity of bile pigment excreted, the rate of hemoglobin turnover has
been calculated to #. 16 to 24 gm. per day under normal conditions.
SECTION XVI — PLATE I 2 BILE PIGMENT METABoLISM Normal and Abnormal
From the quantity of bile pigment excreted, the rate of hemoglobin turnover has
been calculated to #. 16 to 24 gm. per day under normal conditions.
Page 48
Increased production or decreased excretion of biliary pigments leads to
jaundice without bile flow impairment. Hemolytic jaundice is due to
overproduction of bile pigment. It occurs in septicemia, pernicious anemia,
acquired or congenital ...
Increased production or decreased excretion of biliary pigments leads to
jaundice without bile flow impairment. Hemolytic jaundice is due to
overproduction of bile pigment. It occurs in septicemia, pernicious anemia,
acquired or congenital ...
Page 159
... 145 teratoid, 60 cystadenocarcinoma, 136, 147 cystadenoma, 136, 145, 146
cysteine conjugation, 44, 64 cystic disease, liver, 60 pancreas, see fibrocystic
disease cystine deficiency, 77 cytochrome pigment, 88 cytologic smear
examination, ...
... 145 teratoid, 60 cystadenocarcinoma, 136, 147 cystadenoma, 136, 145, 146
cysteine conjugation, 44, 64 cystic disease, liver, 60 pancreas, see fibrocystic
disease cystine deficiency, 77 cytochrome pigment, 88 cytologic smear
examination, ...
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Contents
SECTION | 1 |
plate PAGE NUMBER Num BER 1 Development of Liver and Its Venous System | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute amylase anastomoses Anatomy appear ascites biliary obstruction biliary tract bilirubin biopsy blood capillaries carcinoma cause celiac cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal esophageal varices excretion extrahepatic fibers fibrosis fistula flocculation formation frequently function gallbladder gland glycogen hepatic duct hepatic tests hepatic vein hepatocellular increased infection inferior injury intestinal intrahepatic jaundice Kupffer cells left gastric left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism necrosis nodes normal º º organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules renal result right hepatic artery SECTION septa serum shunt sinusoids sphincter spleen splenic stones superior mesenteric surface surgical tion tumor urine urobilinogen varices vena cava vessels viral hepatitis