The Ciba Collection of Medical Illustrations: pt. 1. Upper digestive tract |
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Page 10
The covering, however, is much thinner than that of the structures of the portal
triads, which explains the easy yielding of the hepatic vein system to
compression (see page 69). The ramifications of the hepatic vein and of the
portal vein cross ...
The covering, however, is much thinner than that of the structures of the portal
triads, which explains the easy yielding of the hepatic vein system to
compression (see page 69). The ramifications of the hepatic vein and of the
portal vein cross ...
Page 83
Subsequently, cellular infiltration of the portal triads takes place, with some
proliferation of perilobular cholangioles and periportal ducts. An inflammatory
reaction ensues, without bacterial infection, probably as a result of a reaction to
the bile ...
Subsequently, cellular infiltration of the portal triads takes place, with some
proliferation of perilobular cholangioles and periportal ducts. An inflammatory
reaction ensues, without bacterial infection, probably as a result of a reaction to
the bile ...
Page 92
If the microorganisms reach the sinusoids, they are . taken up and destroyed by
the Kupffer cells or they pass into the tissue spaces, whence they are drained to
the lymphatics in the portal triads to set up an inflammation within and around the
...
If the microorganisms reach the sinusoids, they are . taken up and destroyed by
the Kupffer cells or they pass into the tissue spaces, whence they are drained to
the lymphatics in the portal triads to set up an inflammation within and around the
...
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Contents
SECTION | 1 |
plate PAGE NUMBER Num BER 1 Development of Liver and Its Venous System | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid acute amylase anastomoses Anatomy appear ascites biliary obstruction biliary tract bilirubin biopsy blood capillaries carcinoma cause celiac cholestasis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated ductules duodenal duodenum enzymes esophageal esophageal varices excretion extrahepatic fibers fibrosis fistula flocculation formation frequently function gallbladder gland glycogen hepatic duct hepatic tests hepatic vein hepatocellular increased infection inferior injury intestinal intrahepatic jaundice Kupffer cells left gastric left hepatic left lobe lesions ligament liver cell plates liver disease lobular lobule lymphatic metabolism necrosis nodes normal º º organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal tracts portal triads portal vein posterior protein regenerative nodules renal result right hepatic artery SECTION septa serum shunt sinusoids sphincter spleen splenic stones superior mesenteric surface surgical tion tumor urine urobilinogen varices vena cava vessels viral hepatitis